Keratosis Pilaris Diet

Keratosis pilaris diet and general information

        Keratosis pilaris is often transmitted as an autosomal dominant. External factors are rarely in question. Keratosis pilaris is common in patients with atopic dermatitis. Sometimes it is part of some genetic diseases (syndromes Noonan, of Fairbanks, in Rombo, Pachyonychia congenital Acrokeratosis verruciformis of Hopf, etc.).. An inflammatory reaction is possible, including scarring (fibrosis).

        The keratosis, also known as hyperkeratosis is thickening of the stratum corneum (the most superficial layer of the epidermis, which contains the protein keratin). This term refer to either outgrowths such as corns, calluses and warts, or skin diseases, which are the main Pilaris keratosis, senile and solar. The latter develops in people exposed for years to a strong ensoleillement. Kratosis pilaris is very common, especially among adolescents. It appears as a small reddish rash, rough as crusts, and most commonly affects the outer surface of the arms and thighs. Unpleasant but benign, keratosis pilaris is treated by application of keratolytic drugs, based on urea or salicylic acid.

        In severe cases, the dermatologist may prescribe vitamin A in very high doses (100 000-200 000 IU) for weeks or months. Please note: Excess vitamin A (the hypervitaminosis A) can cause fatigue, headaches, dizziness and vomiting. At hyperkeratosis, an increase in vitamin A intake through diet is an inadequate measure, since this substance has a keratolytic action. Attention to refined sugars! Until more studies are conducted in this area, anyone who is concerned with his look (and his health) will take the most from complete carbohydrates (cereals, bread and pasta) to the refined food.